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subject: Cva Homeopathic Approach [print this page]


It is defined as a "neurological deficit of cerebro vascular cause that persists beyond 24 hours or is interrupted by death within 24 hours".

The 24-hour limit divides stroke from transient ischemic attack, which is a related syndrome of stroke symptoms that resolve completely within 24 hours.

Classification

Strokes can be classified into two major categories: ischemic and hemorrhagic. Ischemic strokes are those that are due to interruption of the blood supply, while hemorrhagic strokes are the ones which are due to rupture of a blood vessel or an abnormal vascular structure. 80% of strokes are due to ischemia; the remaining are due to hemorrhage

1.Ischemic stroke

There are four reasons why this might happen:

1. Thrombosis (obstruction of a blood vessel by a blood clot forming locally)

2. Embolism (obstruction due to an embolus from elsewhere in the body, see below)

3. general decrease in the blood supply for example in the condition of shock

4. venous thrombosis

Hemorrhagic stroke

Intracranial hemorrhage is the accumulation of blood any where within the skull vault.

The main types of extra axial hemorrhagea are as follows:

1.epidural hemorrhage

2.sub dural hemorrhage

3.sub arachniod hemorrhage

Most of the hemorrhagic stroke syndromes have specific symptoms (e.g. headache, previous head injury). Intra cerebral hemorrhage (ICH) is bleeding directly into the brain tissue, forming a gradually enlarging hematoma

Risk factors

The most important modifiable risk factors for stroke are

1.high blood pressure

2. atrial fibrillation

3.high blood cholesterol levels,

4.diabetes,

5.cigarette smoking

6.heavy alcohol consumption

7.drug use

8.lack ofphysical activity

9. obesity

10.Unhealthy diet

Alcohol use could predispose to ischemic stroke, and intracerebral and subarachnoid hemorrhage via multiple mechanisms.

Signs and symptoms

Stroke symptoms typically start suddenly, over seconds to minutes, and in most cases do not progress further. The symptoms depend on the area of the brain affected. The more extensive the area of brain affected, the more functions that are likely to be lost.

Hemiplegia and muscle weakness of the face

numbness

reduction in sensory or vibratory sensation

altered smell, taste, hearing, or vision (total or partial)

drooping of eyelid (ptosis)and weakness of ocular muscles

decreased reflexes: gag, swallow, pupil reactivity to light

balance problems and nystagmus

altered breathing and heart rate

weakness in sterno cleido mastoid muscle with inability to turn head to one side

weakness in tongue (inability to protrude and/or move from side to side)

If the cerebral cortex is involved, the CNS pathways can again be affected, but also can produce the following symptoms:

aphasia (inability to speak or understand language from involvement of brocas or wernicks area)

apraxia (altered voluntary movements)

visual field defect

memory deficits

disorganized thinking, confusion

anosognosia (persistent denial of the existence of a, usually stroke-related, deficit)

If the cerebellum is involved, the patient may have the following:

trouble walking

altered movement coordination

vertigo and or disequilibrium

DIAGNOSIS:

Signs and symptoms at the onset of the attack

Physical examination

INVESTIGATIONS:

CT scan of the brain

MRI scan of the brain

CSF examination

Positron emission tomography

Complete blood picture

MANAGEMENT:

Avoidance of alcohol

Cessation of the smoking habit

Good nutritious food

Regular physiotherapy exercise

Control of blood pressure

Reduction of weight

Control of blood lipids

HOMOEOPATHIC APPROACH

CAUSTICUM:

Paralysis from exposure to the cold of winter

Face is usually affected

The paralysis remaining after the patient has recovered other wise

Aphonia

Weakness of sphincter vesicae

Ptosis of rheumatic origin

BARYTA CARBONICA:

Paralysis produced by degeneration of coats of blood vessels

Paralysis after apoplexy

Want of stediness

Facial paralysis where tongue is implicated

Suited to the paralysis of old people

ACONITUM NAPELLUS

For the acute cases of paralysis

Numbness and tingling of the affected parts

Facial paralysis accompanied with coldness from exposure to dry cold winds

Paraplegia with tingling

GELSEMIUM:

Complete motor paralysis

Paralysis of the ocular muscles, ptosis

Speech is thick from the paretic conditions of the tongue

Paralysis from the emotions

Aphonia, paralysis of the larynx

by: Gen Wright




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