subject: Forms of HSV Infection [print this page] Author: bcured Author: bcured
Viral infections of the eye that occur after the neonatal period are usually caused by HSV-1. Approximately 300,000 cases of HSV infections of the eye are diagnosed annually in the United States. HSV keratoconjunctivitis is associated with either unilateral or bilateral conjunctivitis, which can be follicular in nature and is followed soon thereafter by preauricular adenopathy. Herpetic keratoconjunctivitis is also associated with photophobia, tearing, eyelid edema, and chemosis, as well as the pathognomonic findings of branching dendritic lesions. Geographic ulcers of the cornea develop in patients with advanced disease. The rate of recurrence parallels that described for herpes labialis. Most frequently, recurrences are unilateral but in a small percentage of cases involve both eyes. Immunocompromised patients, especially organ transplant recipients, are at increased risk for severe HSV infections. These patients may develop progressive disease involving the respiratory tract, esophagus, or gastrointestinal tract. The severe nature of progressive disease in these patients appears to be directly related to the amount and duration of immunosuppressive therapy. Esophagitis occurs commonly in immunocompromised patients and can be caused by HSV, cytomegalovirus, or Candida albicans. Acyclovir-resistant HSV disease can occur in immunocompromised patients and can be progressive, especially in those with AIDS. Encephalitis is one of the most devastating of all HSV infections; HSV is considered the most common cause of sporadic, fatal encephalitis. The manifestations of HSV encephalitis in older children and adults are indicative of the areas of the brain affected. These manifestations include primarily focal encephalitis that is associated with fever, altered consciousness, bizarre behavior, disordered mentation, and localized neurological findings. Clinical signs and symptoms reflect localized temporal lobe disease. No signs are pathognomonic for HSV encephalitis; however, a progressively deteriorating level of consciousness in association with fever, an abnormal CSF profile, and focal neurological findings in the absence of other causes should make this disease highly suspect. Diagnostic evaluations should be initiated immediately, since other treatable diseases mimic HSV encephalitis. The mortality among untreated patients exceeds 70%, and only 2.5% of patients who survive regain normal neurological function. Standard neurodiagnostic procedures are used in the evaluation of patients with suspected HSV encephalitis and include CSF examination, electroencephalography, and scanning procedures such as CT or MRI. Characteristic abnormalities of the CSF include elevated levels of WBCs, RBCs, and protein. Spike and slow wave activity are generally localized to the temporal lobe on electroencephalograms. A burst suppression pattern is characteristic of HSV encephalitis. Imaging will allow for localization of disease to the temporal lobe. Early after the onset of disease, only evidence of edema is detectable, if at all. This finding is followed by evidence of hemorrhage and midline shift in the cortical structures. In addition to the brain, HSV can involve virtually all anatomic areas of the nervous system, causing manifestations such as meningitis, myelitis, and radiculitis. HSV has been isolated from the respiratory tracts of adults with adult respiratory distress syndrome and acute-onset bron chospasm. Both conditions are associated with increased mortality and morbidity. Acyclovir is the most popular prescribed cure for herpes viral dispersion. However, the emergence of aciclovir-resistant virus strains has created the need for the development of new effective antiviral agents. New anti-herpetic chemical drug compounds have been identified as possible cures for herpes, but they have significant adverse effects when consumed and HSV has again has developed drug resistance to these new compounds. HSVCurative is used specifically to treat HSV1 and HSV2 infections and acts as a curative agent against both these strains of herpes. It exhibits a pronounced anti-herpetic activity against HSV1 and HSV2 and, unlike other cures for herpes, actually kills these viruses upon exposure regardless of location on the body. HSVCurative contains certified organic medicinal plant extracts and antiviral essential oils which cause cellular death to HSV1 and HSV2 in scientific studies. HSVCurative has a rather remarkable array of pharmacological and biochemical properties which inactivate and destroy the herpes virus for unprecedented results compared to other cures for herpes. Very recent studies using advanced DNA techniques have shown that HSVCurative extracts indeed destroy HSV1 and HSV2, the common causes of oral and genital herpes. A cure of these viruses has been elusive until recently. To learn more, please go to http://www.bcured.net.About the Author:
staff of Nature Power Company, which is a network company dedicated to promoting customers' websites and developing softwares. You can go to the following websites to learn more about our natural organic products. http://www.bcured.nethttp://www.naturespharma.org
